01 Overview

Stanford Chemicals sells only the highest quality Dihydromyricetin (DHM) and stand behind our product. Our DHM is manufactured in the United States, and is rigorously tested for purity and effectiveness.

02 Product Description

Introduction of Dihydromyricetin:

Product Name Dihydromyricetin
CAS Registry Number 27200-12-0
Molecular Formula C15H12o8
Molecular Weight 320.25
Purity 98%HPLC
Package 20mg-1g
Storge In sealed, cool and dry condition
Category Standard

Stanford Chemicals makes the most effective intoxication blocker and hangover cure available to the general population. We believe responsible use of DHM can greatly reduce the risks associated with alcohol consumption and provide relief for the next day’s hangover.

Why Stanford Chemicals?

1. Original fermentation technology
Stanford Chemicals’ original manufacture method results in high purity. Our sodium hyaluronate not only complies with public standard but also meets the more strict specification of our own.
2. Non-animal origin guaranteed
Stanford Chemicals is committed to ensuring non-animal origin for all raw materials in the manufacture of sodium hyaluronate.
3. Certifications and Compliance
Stanford Chemicals is managed strictly in accordance with GMP and ISO 9001 quality management system to ensure the reliability and stability of products.
4 Made In the USA
Our Dihydromyricetin is manufactured right here in the USA, in a cGMP certified facility.

Applications of Dihydromyricetin:

Dihydromyricetin (DHM) is a natural extract of the Oriental Raisin Tree. Dihydromyricetin will change your relationship with alcohol forever. No product is more proven to prevent all hangover symptoms. You will wake-up not knowing you drank the night before. Research has shown that it reduces symptoms of intoxication, reduces damage done to the liver, and prevents hangovers. Dihydromyricetin powder achieves these effects by both improving the livers’ ability to metabolize alcohol and by blocking alcohol from affecting the brain’s GABA receptors.

1 PREVENT HANGOVERS – Dihydromyricetin (DHM) is called the ‘antidote to alcohol’ because of how effectively it prevents feelings of intoxication, headbanging hangovers and other unpleasant alcohol withdrawal symptoms.
2 PROTECT YOUR LIVER AND BRAIN – Research shows Dihydromyricetin reduces alcohol-induced damage to the liver and brain.
3 NO MORE WASTED DAYS – Wake up feeling more refreshed and alert after a night out. Enjoy yourself at night and be productive the next day.

antialcoholic drug

Dihydromyricetin (DHM or DMY), sometimes called Ampelopsin or Ampeloptin, is a natural antioxidant flavonoid from the Chinese vine tea plants (Ampelopsis grossedentata).

As an active herbal ingredient of the extract, it shows very promising results amongst hangover cures and has a significant protective effect against alcohol-induced liver damage.
Dihydromyricetin can work as an active free radical scavenger. The percentage inhibition of DPPH could reach 73.3%-91.5% and with the IC50 value of 5.4 mmol/L. It markedly attenuates the activity of superoxide anion generation. It also has the strong inhibitory action of Staphylococcus aureus, Bacillus subtilis and Aspergillus flavus etc. New applications have been confirmed recently that DHM can reduce the levels of blood sugar and blood fat; anti-inflammation; effective restraint to cell proliferation of some tumor cells.

hovenia fruit VS vine tea

Related Articles of Dihydromyricetin Extracts Powder:

Everything You Need to Know About Dihydromyricetin

The Miraculous Effect of Dihydromyricetin

Dihydromyricetin – “Soft Gold in Flavonoids”

Can Dihydromyricetin Relieve Alcohol?

List of Dihydromyricetin Benefits

03 Questions & Answers

  1. 1
    What is DHM?

    Dihydromyricetin (DHM) is an organic compound naturally found in the Raisin Tree. It’s often called by its Latin classification Hovenia Dulcis, and is found all over Asia. As a deciduous tree, it grows up to 12m tall, and bears a berry-like fruit that tastes similar to raisins, from which it gets its name. The molecule we know as Dihydromyricetin is also called Ampelopsin, and is extracted using an ethanol-based solution from the bark of the Hovenia Dulcis tree, and minimally processed before it’s dehydrated and turned into a powder ready to be purified.

  2. 2
    Should I take it before or after drinking?

    When you take dihydromyricetin really depends on what you’re trying to accomplish.  So, for example, researchers found that taking a relatively large dose before drinking it reduced the circulating levels of alcohol in the blood.  For most people however, we recommend taking it immediately after drinking which works very well to reduce the hangover symptoms usually felt the next day and stops the GABA rebound effect that will often wake you up in the middle of the night or give you anxiety the next day.

  3. 3
    Does Dihydromyricetin work?

    Yes.  For both reducing intoxication, and for reducing the effects of a hangover and supporting the liver, there is ample scientific evidence.  The question really is, at what dose does it work?  And for that the answer is slightly different and varies by the number of drinks you’ve had, their alcohol content, whether you’ve eaten and so on.  It also depends on what you’re looking to achieve with DHM, since reducing intoxication requires significantly higher doses, than when taken after drinking to eliminate hangovers.

  4. 4
    Will DHM stop me getting drunk?

    Not really.  DHM will reduce the impact alcohol has on your central nervous system (by down-regulating GABA transmission) which means you may be more co-ordinated and less likely to have slurred speech if you take Dihydromyricetin when consuming alcohol.

  5. 5
    Will it sober me up faster?

    Theoretically, yes.  Doses to reduce circulating alcohol content need to be relatively large — in the 3g+ range for humans — and you still won’t be safe to drive no matter what, and doing so would be illegal.  Large doses of the supplement will indeed make you feel less drunk, but its not certain whether this is because the anti-alcohol effect is strong enough by itself or whether the additional reduction in the neurotransmitter GABA provides an additive effect.

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